Across TCGA pan-cancer cohorts, VN1R32P RNA differs between tumor and matched normal tissue in 1 of 18 cancer types tested, making tumor–normal expression one of VN1R32P’s most consistent transcriptional readouts.
The strongest signal is observed in liver hepatocellular carcinoma (LIHC), where VN1R32P RNA is more highly expressed in tumor relative to normal tissue. In most cancer types VN1R32P is over-expressed in tumor.
LIHC are the cancer types where VN1R32P tumor–normal differential expression is most reproducible.