Q-omics provides the consensus-scored UQCR10P1 profile across patient tissues and cancer cell-line models. UQCR10P1 expression is associated with patient survival in 16 of 34 cancer types, with the highest sampling consensus in KIRP. Among the 18 cancer types available for tumor–normal comparison, UQCR10P1 is differentially expressed in 3, with the highest sampling consensus in THCA. Additionally, UQCR10P1 RNA expression shows 6,059 significant protein co-abundance associations, with the highest sampling consensus in OV. Together, these results highlight KIRP, THCA, and OV as cancer lineages where UQCR10P1 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for UQCR10P1 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes UQCR10P1 survival associations across molecular data types. UQCR10P1 RNA expression shows survival associations in the most cancer types (16). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible UQCR10P1 RNA expression–survival associations across cancer types. High UQCR10P1 expression shows unfavorable associations in KIRP and LIHC, but favorable associations in ACC, BLCA, PAAD and LGG. The KIRP Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify KIRP as the clearest survival context for UQCR10P1 RNA expression.
This table summarizes UQCR10P1 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 3. The strongest signals are observed in THCA for RNA.
This table ranks reproducible tumor–normal expression differences for UQCR10P1. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. UQCR10P1 shows lower tumor expression in THCA and higher tumor expression in STAD and LIHC. The THCA box plot shows higher UQCR10P1 RNA expression in normal versus tumor tissue (log2 FC = −0.104, t-test p = .015).
This table shows molecular features associated with UQCR10P1 in patient tissues and cancer cell lines. In patient samples, UQCR10P1 shows the broadest associations at the RNA and protein expression levels, with OV recurring as the lineage with the largest associated feature set.
