Across TCGA pan-cancer cohorts, SRP68P2 RNA differs between tumor and matched normal tissue in 2 of 18 cancer types tested, making tumor–normal expression one of SRP68P2’s most consistent transcriptional readouts.

The strongest signal is observed in breast invasive carcinoma (BRCA), where SRP68P2 RNA is repressed in tumor relative to normal tissue. In most cancer types SRP68P2 is over-expressed in tumor, although a few such as BRCA show the opposite, repressed pattern.

BRCA and KIRC are the cancer types where SRP68P2 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in SRP68P2 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
BRCA	Female	II,III,IV	−0.162	.047	2	view →
KIRC	All	II,III,IV	+0.091	.043	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 2 strongest of 2 lineages.

Exploration

Same gene, other analysis: SRP68P2 RNA expression & survival · all SRP68P2 data types