SNHG30

Across TCGA pan-cancer cohorts, SNHG30 RNA differs between tumor and matched normal tissue in 14 of 18 cancer types tested, making tumor–normal expression one of SNHG30’s most consistent transcriptional readouts.

The strongest signal is observed in kidney chromophobe (KICH), where SNHG30 RNA is repressed in tumor relative to normal tissue. In most cancer types SNHG30 is over-expressed in tumor, although a few such as KICH and THCA show the opposite, repressed pattern.

KICH, LIHC, and THCA are the cancer types where SNHG30 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in SNHG30 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KICH	Male	IV	−2.316	<.001	11	view →
LIHC	Male	II,III,IV	+1.241	<.001	9	view →
THCA	Male	III,IV	−1.094	<.001	9	view →
HNSC	Male	IV	+1.317	<.001	8	view →
KIRC	Male	II,III,IV	−0.768	<.001	8	view →
BLCA	All	All	+0.672	.010	6	view →
BRCA	Female	II,III,IV	−0.596	<.001	6	view →
LUSC	Female	All	+0.931	<.001	5	view →
LUAD	All	II,III,IV	−0.541	.001	4	view →
COAD	All	All	+0.329	.012	4	view →
CHOL	Male	All	+2.035	<.001	3	view →
STAD	All	II,III,IV	+0.607	.007	3	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 12 strongest of 14 lineages.

SNHG30–KICH

Open the KICH breakdown →