Across TCGA pan-cancer cohorts, SLC30A2 Mutation is linked to patient survival in 3 of 34 cancer types, making it a survival-associated SLC30A2 data layer compared with 23 for mass-spec protein and 2 for mass-spec protein.
The strongest signal is observed in lung adenocarcinoma (LUAD), where higher SLC30A2 Mutation is associated with worse overall survival. In most high-consensus cancer types, elevated SLC30A2 expression acts as an unfavorable survival marker.
LUAD, PRAD, and COAD are the cancer types where SLC30A2 Mutation most reproducibly stratifies survival.