Across TCGA pan-cancer cohorts, SLC26A9 Mutation is linked to patient survival in 4 of 34 cancer types, making it a survival-associated SLC26A9 data layer compared with 23 for mass-spec protein.
The strongest signal is observed in uterine corpus endometrial carcinoma (UCEC), where higher SLC26A9 Mutation is associated with better disease-free survival. In most high-consensus cancer types, elevated SLC26A9 expression acts as an unfavorable survival marker, although some lineages such as UCEC show a favorable association.
UCEC, UVM, and PRAD are the cancer types where SLC26A9 Mutation most reproducibly stratifies survival.