Across TCGA pan-cancer cohorts, SLC26A11 Mutation is linked to patient survival in 2 of 34 cancer types, making it a survival-associated SLC26A11 data layer compared with 26 for mass-spec protein and 2 for mass-spec protein.
The strongest signal is observed in rectum adenocarcinoma (READ), where higher SLC26A11 Mutation is associated with worse overall survival. In most high-consensus cancer types, elevated SLC26A11 expression acts as an unfavorable survival marker.
READ and SKCM are the cancer types where SLC26A11 Mutation most reproducibly stratifies survival.