Across TCGA pan-cancer cohorts, SELENOM Mutation is linked to patient survival in 3 of 34 cancer types, making it a survival-associated SELENOM data layer compared with 25 for mass-spec protein and 6 for mass-spec protein.

The strongest signal is observed in prostate adenocarcinoma (PRAD), where higher SELENOM Mutation is associated with worse disease-free survival. In most high-consensus cancer types, elevated SELENOM expression acts as an unfavorable survival marker.

PRAD, LUSC, and UCEC are the cancer types where SELENOM Mutation most reproducibly stratifies survival.

Mutation survival associations by lineage

Ranked by sampling consensus. AUC1 and AUC2 indicate survival in the high- and low-expression groups, respectively; the lower AUC marks the poorer-surviving group. p-values are from the log-rank test.

Lineage	Measure	Split	Stage	AUC1 high	AUC2 low	p	Sampling consensus
PRAD	DFS	Median	All	0.085	0.774	<.001	6	view →
LUSC	OS	Median	All	0.192	0.724	.040	6	view →
UCEC	OS	Median	IV	0.231	0.592	.036	6	view →

Pink = unfavorable, green = favorable. Showing the 3 strongest of 3 lineages.

Exploration

Same gene, other analysis: SELENOM tumor-vs-normal RNA expression · all SELENOM data types