SCAANT1

Across TCGA pan-cancer cohorts, SCAANT1 RNA differs between tumor and matched normal tissue in 7 of 18 cancer types tested, making tumor–normal expression one of SCAANT1’s most consistent transcriptional readouts.

The strongest signal is observed in kidney chromophobe (KICH), where SCAANT1 RNA is repressed in tumor relative to normal tissue. In most cancer types SCAANT1 is over-expressed in tumor, although a few such as KICH and READ show the opposite, repressed pattern.

KICH, STAD, and HNSC are the cancer types where SCAANT1 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in SCAANT1 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KICH	Female	II,III,IV	−1.074	<.001	6	view →
STAD	Male	II,III,IV	+0.574	.001	6	view →
HNSC	Male	III,IV	+0.425	.007	5	view →
LIHC	All	All	+0.319	.003	4	view →
PRAD	All	All	+0.333	.011	2	view →
LUSC	Male	III,IV	+0.692	.024	1	view →
READ	All	All	−0.388	.029	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 7 strongest of 7 lineages.

SCAANT1–KICH

Open the KICH breakdown →