SALL4P6

associated omics data
spalt like transcription factor 4 pseudogene 6Genealiases: []

Q-omics provides the consensus-scored SALL4P6 profile across patient tissues and cancer cell-line models. SALL4P6 expression is associated with patient survival in 17 of 34 cancer types, with the highest sampling consensus in COAD. Among the 18 cancer types available for tumor–normal comparison, SALL4P6 is differentially expressed in 3, with the highest sampling consensus in KIRP. Additionally, SALL4P6 RNA expression shows 6,019 significant pathway-activity associations, with the highest sampling consensus in STAD. Together, these results highlight COAD, KIRP, and STAD as cancer lineages where SALL4P6 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.

Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.

Survival associations

This table summarizes SALL4P6 survival associations across molecular data types. SALL4P6 RNA expression shows survival associations in the most cancer types (17). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
SALL4P6 data typeSurvival analysisLineage consensusLineage of highest sampling consensus
RNAKaplan–Meier17COAD (75)view →
This table ranks reproducible SALL4P6 RNA expression–survival associations across cancer types. High SALL4P6 expression shows unfavorable associations in COAD, LUSC, READ, LIHC and ACC, but favorable associations in KIRC. The COAD Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify COAD as the clearest survival context for SALL4P6 RNA expression.
LineageMeasureSplitStageAUC1
high
AUC2
low
pSampling consensus
COADOSTertileIV0.1110.658<.00175view →
LUSCOSTertileIII,IV0.1370.672<.00154view →
KIRCDFSMedianII,III,IV0.7790.645.00854view →
READDFSTertileAll0.3150.866<.00151view →
LIHCOSTertileAll0.5300.783.00545view →
ACCDFSTertileAll0.1840.676<.00136view →
Pink = unfavorable, green = favorable. all 17 lineages →

SALL4P6-COAD (OS)

Kaplan–Meier survival curve for SALL4P6 RNA expression in COAD: high vs low expression groups.

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Tumor vs Normal expression

This table summarizes SALL4P6 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 3. The strongest signals are observed in KIRP for RNA.
SALL4P6 data typeExpression analysisLineage consensusLineage of highest sampling consensus
RNABox plot3KIRP (3)view →
This table ranks reproducible tumor–normal expression differences for SALL4P6. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. SALL4P6 shows lower tumor expression in COAD and higher tumor expression in KIRP and KIRC. The KIRP box plot shows higher SALL4P6 RNA expression in tumor versus normal tissue (log2 FC = +0.006, t-test p = .010).
LineageGenderStageFold-changepSampling consensus
KIRPAllAll+0.006.0103view →
KIRCMaleAll+0.016.0102view →
COADAllAll−0.005.0471view →
Green = repressed in tumor. all 3 lineages →

SALL4P6-KIRP

Tumor-vs-normal expression box plot for SALL4P6 in KIRP.

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Cross-omics associations

This table shows molecular features associated with SALL4P6 in patient tissues and cancer cell lines. In patient samples, SALL4P6 shows the broadest associations at the RNA and protein expression levels, with STAD recurring as the lineage with the largest associated feature set.
Associated data typeStrength (# associated data)Lineage of highest associated data
RNA
Function (RNA)6,019STAD (4059)view →
RNA2,604KIRC (547)view →