RSL24D1P11

Across TCGA pan-cancer cohorts, RSL24D1P11 RNA differs between tumor and matched normal tissue in 8 of 18 cancer types tested, making tumor–normal expression one of RSL24D1P11’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where RSL24D1P11 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types RSL24D1P11 is over-expressed in tumor, although a few such as BRCA and UCEC show the opposite, repressed pattern.

KIRC, BRCA, and UCEC are the cancer types where RSL24D1P11 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in RSL24D1P11 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	Male	III,IV	+0.242	<.001	9	view →
BRCA	All	III,IV	−0.301	<.001	6	view →
UCEC	All	All	−0.356	.002	4	view →
LUSC	Female	All	−0.184	.002	4	view →
READ	All	All	−0.207	.012	2	view →
KICH	Female	All	−0.129	.022	2	view →
KIRP	All	IV	+0.110	.030	2	view →
BLCA	All	IV	−0.220	.033	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 8 strongest of 8 lineages.

RSL24D1P11–KIRC

Open the KIRC breakdown →