RPSAP16

Across TCGA pan-cancer cohorts, RPSAP16 RNA differs between tumor and matched normal tissue in 7 of 18 cancer types tested, making tumor–normal expression one of RPSAP16’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where RPSAP16 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types RPSAP16 is over-expressed in tumor, although a few such as THCA show the opposite, repressed pattern.

KIRC, BRCA, and CHOL are the cancer types where RPSAP16 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in RPSAP16 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	All	All	+0.047	<.001	6	view →
BRCA	All	All	+0.106	.025	4	view →
CHOL	Male	All	+0.131	.014	2	view →
PRAD	All	All	+0.095	.002	2	view →
THCA	Female	II,III,IV	−0.073	.039	2	view →
KIRP	All	All	+0.082	.016	1	view →
LIHC	Female	II,III,IV	+0.065	.038	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 7 strongest of 7 lineages.

RPSAP16–KIRC

Open the KIRC breakdown →