RPS24P17

Across TCGA pan-cancer cohorts, RPS24P17 RNA differs between tumor and matched normal tissue in 7 of 18 cancer types tested, making tumor–normal expression one of RPS24P17’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where RPS24P17 RNA is repressed in tumor relative to normal tissue. In most cancer types RPS24P17 is over-expressed in tumor, although a few such as KIRC and KIRP show the opposite, repressed pattern.

KIRC, KIRP, and KICH are the cancer types where RPS24P17 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in RPS24P17 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	Female	III,IV	−2.590	<.001	12	view →
KIRP	Female	II,III,IV	−3.630	<.001	11	view →
KICH	Female	All	−2.512	<.001	7	view →
COAD	All	III,IV	+0.634	.003	7	view →
LUAD	Female	All	+0.292	.002	2	view →
THCA	All	III,IV	+0.195	.025	2	view →
CHOL	All	All	+0.254	.011	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 7 strongest of 7 lineages.

RPS24P17–KIRC

Open the KIRC breakdown →