Q-omics provides the consensus-scored RPS23P3 profile across patient tissues and cancer cell-line models. RPS23P3 expression is associated with patient survival in 13 of 34 cancer types, with the highest sampling consensus in KICH. Among the 18 cancer types available for tumor–normal comparison, RPS23P3 is differentially expressed in 6, with the highest sampling consensus in KIRC. Additionally, RPS23P3 RNA expression shows 10,229 significant gene co-expression associations, with the highest sampling consensus in THYM. Together, these results highlight KICH, KIRC, and THYM as cancer lineages where RPS23P3 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for RPS23P3 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes RPS23P3 survival associations across molecular data types. RPS23P3 RNA expression shows survival associations in the most cancer types (13). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible RPS23P3 RNA expression–survival associations across cancer types. High RPS23P3 expression shows unfavorable associations in KICH, CHOL, STAD, ACC, PCPG and LIHC. The KICH Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify KICH as the clearest survival context for RPS23P3 RNA expression.
This table summarizes RPS23P3 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 6. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for RPS23P3. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. RPS23P3 shows higher tumor expression in KIRC, LUSC, BRCA, LUAD, LIHC and STAD. The KIRC box plot shows higher RPS23P3 RNA expression in tumor versus normal tissue (log2 FC = +0.070, t-test p < 0.001).
This table shows molecular features associated with RPS23P3 in patient tissues and cancer cell lines. In patient samples, RPS23P3 shows the broadest associations at the RNA and protein expression levels, with THYM recurring as the lineage with the largest associated feature set.
