Q-omics provides the consensus-scored RPS15AP11 profile across patient tissues and cancer cell-line models. RPS15AP11 expression is associated with patient survival in 19 of 34 cancer types, with the highest sampling consensus in KIRP. Among the 18 cancer types available for tumor–normal comparison, RPS15AP11 is differentially expressed in 8, with the highest sampling consensus in KIRC. Additionally, RPS15AP11 RNA expression shows 13,657 significant protein co-abundance associations, with the highest sampling consensus in GBM. Together, these results highlight KIRP, KIRC, and GBM as cancer lineages where RPS15AP11 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for RPS15AP11 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes RPS15AP11 survival associations across molecular data types. RPS15AP11 RNA expression shows survival associations in the most cancer types (19). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible RPS15AP11 RNA expression–survival associations across cancer types. High RPS15AP11 expression shows unfavorable associations in KIRP, LUAD, ACC, LIHC and MESO, but favorable associations in LUSC. The KIRP Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p = .002). Together, the overview and detailed table identify KIRP as the clearest survival context for RPS15AP11 RNA expression.
This table summarizes RPS15AP11 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 8. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for RPS15AP11. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. RPS15AP11 shows lower tumor expression in READ and COAD and higher tumor expression in KIRC, KIRP, PRAD and CHOL. The KIRC box plot shows higher RPS15AP11 RNA expression in tumor versus normal tissue (log2 FC = +0.707, t-test p < 0.001).
This table shows molecular features associated with RPS15AP11 in patient tissues and cancer cell lines. In patient samples, RPS15AP11 shows the broadest associations at the RNA and protein expression levels, with GBM recurring as the lineage with the largest associated feature set.
