RN7SL393P

Across TCGA pan-cancer cohorts, RN7SL393P RNA differs between tumor and matched normal tissue in 5 of 18 cancer types tested, making tumor–normal expression one of RN7SL393P’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where RN7SL393P RNA is repressed in tumor relative to normal tissue. In most cancer types RN7SL393P is over-expressed in tumor, although a few such as KIRC and KICH show the opposite, repressed pattern.

KIRC, KICH, and LUSC are the cancer types where RN7SL393P tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in RN7SL393P RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	All	III,IV	−0.111	.001	9	view →
KICH	Female	III,IV	−0.226	<.001	4	view →
LUSC	All	All	+0.197	.001	3	view →
BRCA	Female	All	+0.178	.036	2	view →
HNSC	All	II,III,IV	+0.078	.024	2	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 5 strongest of 5 lineages.

RN7SL393P–KIRC

Open the KIRC breakdown →