Q-omics provides the consensus-scored RN7SL316P profile across patient tissues and cancer cell-line models. RN7SL316P expression is associated with patient survival in 19 of 34 cancer types, with the highest sampling consensus in KIRC. Among the 18 cancer types available for tumor–normal comparison, RN7SL316P is differentially expressed in 5, with the highest sampling consensus in KIRP. Additionally, RN7SL316P RNA expression shows 12,506 significant gene co-expression associations, with the highest sampling consensus in SARC. Together, these results highlight KIRC, KIRP, and SARC as cancer lineages where RN7SL316P shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for RN7SL316P — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes RN7SL316P survival associations across molecular data types. RN7SL316P RNA expression shows survival associations in the most cancer types (19). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible RN7SL316P RNA expression–survival associations across cancer types. High RN7SL316P expression shows unfavorable associations in KIRC, KICH, ACC, UCEC and THCA, but favorable associations in KIRP. The KIRC Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify KIRC as the clearest survival context for RN7SL316P RNA expression.
This table summarizes RN7SL316P tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 5. The strongest signals are observed in KIRP for RNA.
This table ranks reproducible tumor–normal expression differences for RN7SL316P. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. RN7SL316P shows higher tumor expression in KIRP, LUSC, LUAD, CHOL and KIRC. The KIRP box plot shows higher RN7SL316P RNA expression in tumor versus normal tissue (log2 FC = +0.317, t-test p < 0.001).
This table shows molecular features associated with RN7SL316P in patient tissues and cancer cell lines. In patient samples, RN7SL316P shows the broadest associations at the RNA and protein expression levels, with SARC recurring as the lineage with the largest associated feature set.
