Across TCGA pan-cancer cohorts, RAD52P1 RNA differs between tumor and matched normal tissue in 1 of 18 cancer types tested, making tumor–normal expression one of RAD52P1’s most consistent transcriptional readouts.
The strongest signal is observed in kidney renal papillary cell carcinoma (KIRP), where RAD52P1 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types RAD52P1 is over-expressed in tumor.
KIRP are the cancer types where RAD52P1 tumor–normal differential expression is most reproducible.