RAB1AP1

associated omics data
RAB1A pseudogene 1Genealiases: []

Q-omics provides the consensus-scored RAB1AP1 profile across patient tissues and cancer cell-line models. RAB1AP1 expression is associated with patient survival in 24 of 34 cancer types, with the highest sampling consensus in KIRC. Among the 18 cancer types available for tumor–normal comparison, RAB1AP1 is differentially expressed in 12, with the highest sampling consensus in HNSC. Additionally, RAB1AP1 RNA expression shows 11,853 significant gene co-expression associations, with the highest sampling consensus in KIRP. Together, these results highlight KIRC, HNSC, and KIRP as cancer lineages where RAB1AP1 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.

Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.

Survival associations

This table summarizes RAB1AP1 survival associations across molecular data types. RAB1AP1 RNA expression shows survival associations in the most cancer types (24). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
RAB1AP1 data typeSurvival analysisLineage consensusLineage of highest sampling consensus
RNAKaplan–Meier24KIRC (95)view →
This table ranks reproducible RAB1AP1 RNA expression–survival associations across cancer types. High RAB1AP1 expression shows unfavorable associations in KIRC, UVM, ACC, LGG and KIRP, but favorable associations in SKCM. The KIRC Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify KIRC as the clearest survival context for RAB1AP1 RNA expression.
LineageMeasureSplitStageAUC1
high
AUC2
low
pSampling consensus
KIRCDFSQuartileAll0.7270.851<.00195view →
UVMOSTertileAll0.4750.822<.00168view →
ACCOSMedianAll0.4430.755<.00157view →
LGGOSMedianAll0.7550.862.00137view →
SKCMOSQuartileAll0.8450.738.00134view →
KIRPDFSMedianIII,IV0.1550.805.00332view →
Pink = unfavorable, green = favorable. all 24 lineages →

RAB1AP1-KIRC (DFS)

Kaplan–Meier survival curve for RAB1AP1 RNA expression in KIRC: high vs low expression groups.

Explore this curve interactively →

Tumor vs Normal expression

This table summarizes RAB1AP1 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 12. The strongest signals are observed in HNSC for RNA.
RAB1AP1 data typeExpression analysisLineage consensusLineage of highest sampling consensus
RNABox plot12HNSC (10)view →
This table ranks reproducible tumor–normal expression differences for RAB1AP1. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. RAB1AP1 shows lower tumor expression in KICH and higher tumor expression in HNSC, STAD, BRCA, COAD and LUSC. The HNSC box plot shows higher RAB1AP1 RNA expression in tumor versus normal tissue (log2 FC = +0.267, t-test p < 0.001).
LineageGenderStageFold-changepSampling consensus
HNSCFemaleIII,IV+0.267<.00110view →
KICHAllAll−0.100<.0018view →
STADAllAll+0.344.0024view →
BRCAAllAll+0.240<.0014view →
COADAllII,III,IV+0.192.0074view →
LUSCAllAll+0.192.0063view →
Green = repressed in tumor. all 12 lineages →

RAB1AP1-HNSC

Tumor-vs-normal expression box plot for RAB1AP1 in HNSC.

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Cross-omics associations

This table shows molecular features associated with RAB1AP1 in patient tissues and cancer cell lines. In patient samples, RAB1AP1 shows the broadest associations at the RNA and protein expression levels, with KIRP recurring as the lineage with the largest associated feature set.
Associated data typeStrength (# associated data)Lineage of highest associated data
RNA
RNA11,853KIRP (3213)view →
Protein (mass-spec)9,866LSCC (2063)view →