peptidylprolyl isomerase H pseudogene 1Genealiases: []
Q-omics provides the consensus-scored PPIHP1 profile across patient tissues and cancer cell-line models. PPIHP1 expression is associated with patient survival in 21 of 34 cancer types, with the highest sampling consensus in LUAD. Among the 18 cancer types available for tumor–normal comparison, PPIHP1 is differentially expressed in 7, with the highest sampling consensus in COAD. Additionally, PPIHP1 RNA expression shows 11,502 significant gene co-expression associations, with the highest sampling consensus in KIRP. Together, these results highlight LUAD, COAD, and KIRP as cancer lineages where PPIHP1 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for PPIHP1 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes PPIHP1 survival associations across molecular data types. PPIHP1 RNA expression shows survival associations in the most cancer types (21). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible PPIHP1 RNA expression–survival associations across cancer types. High PPIHP1 expression shows unfavorable associations in MESO and READ, but favorable associations in LUAD, THYM, HNSC and SKCM. The LUAD Kaplan–Meier curve shows clear separation, with the low-expression group declining faster, consistent with the favorable association (log-rank p = .001). Together, the overview and detailed table identify LUAD as the clearest survival context for PPIHP1 RNA expression.
This table summarizes PPIHP1 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 7. The strongest signals are observed in LIHC for RNA.
This table ranks reproducible tumor–normal expression differences for PPIHP1. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. PPIHP1 shows higher tumor expression in COAD, LIHC, CHOL, LUSC, PAAD and LUAD. The COAD box plot shows higher PPIHP1 RNA expression in tumor versus normal tissue (log2 FC = +0.358, t-test p = .001).
This table shows molecular features associated with PPIHP1 in patient tissues and cancer cell lines. In patient samples, PPIHP1 shows the broadest associations at the RNA and protein expression levels, with KIRP recurring as the lineage with the largest associated feature set.
