OR51B2

RNA expression — cross-omics
Cross-omicsRNA → FUNCTION-RNACell-linePairwise association · TCGA cohorts

Across TCGA cell cohorts, OR51B2 RNA expression is significantly associated with the go_rna of many other GO terms, with 127 significant associations in total. CNS shows the largest number of these associations.

The most reproducible OR51B2-associated GO terms across cancer lineages are Negative regulation of intracellular transport, Carbon dioxide transport, and Neuron fate commitment. Each is linked with OR51B2 in more than 2 cancer types. Because this analysis shows association rather than direction, both OR51B2-to-partner and partner-to-OR51B2 results are reported.

Each partner links to its own Q-omics profile. The box plot shows the strongest example, Negative regulation of intracellular transport grouped by OR51B2-low versus OR51B2-high in URINARY_TRACT.

RNA expression associated GO terms by consensus

Ranked by combined sampling and lineage consensus. X-score (OR51B2→partner) and Y-score (partner→OR51B2) are standardized regression coefficients; both directions are reported because the association is undirected. p-values are from the association test.
LineagePartner GO termX-scoreY-scorep(X)p(Y)Sampling consensusLineage consensus
URINARY_TRACTNegative regulation of intracellular transport →+0.068+0.124.001.00533
CNSCarbon dioxide transport →+0.947+0.200<.001.00233
CNSNeuron fate commitment →-0.115-0.272<.001.00333
URINARY_TRACTPositive regulation of DNA-templated transcription, elongation →+0.061+0.069<.001.00532
BLOOD_MyelomaRegulation of PERK-mediated unfolded protein response →+0.106+0.143<.001.00532
BLOOD_MyelomaNegative regulation of PERK-mediated unfolded protein response →+0.123+0.139<.001.00732
Each partner links to its Q-omics profile. Showing the 6 strongest of 127 associations by consensus.

Negative regulation of intracellular transport by OR51B2 expression — URINARY_TRACT

Box plot of Negative regulation of intracellular transport in OR51B2-low vs OR51B2-high samples in URINARY_TRACT.

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