olfactory receptor family 13 subfamily K member 1 pseudogeneGenealiases: []
Q-omics provides the consensus-scored OR13K1P profile across patient tissues and cancer cell-line models. OR13K1P expression is associated with patient survival in 14 of 34 cancer types, with the highest sampling consensus in UVM. Among the 18 cancer types available for tumor–normal comparison, OR13K1P is differentially expressed in 8, with the highest sampling consensus in LUAD. Additionally, OR13K1P RNA expression shows 9,063 significant protein co-abundance associations, with the highest sampling consensus in HNSC. Together, these results highlight UVM, LUAD, and HNSC as cancer lineages where OR13K1P shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for OR13K1P — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes OR13K1P survival associations across molecular data types. OR13K1P RNA expression shows survival associations in the most cancer types (14). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible OR13K1P RNA expression–survival associations across cancer types. High OR13K1P expression shows unfavorable associations in UVM, HNSC, ACC, CESC, KIRP and LIHC. The UVM Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify UVM as the clearest survival context for OR13K1P RNA expression.
This table summarizes OR13K1P tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 8. The strongest signals are observed in LUAD for RNA.
This table ranks reproducible tumor–normal expression differences for OR13K1P. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. OR13K1P shows higher tumor expression in LUAD, LUSC, HNSC, BRCA, ESCA and STAD. The LUAD box plot shows higher OR13K1P RNA expression in tumor versus normal tissue (log2 FC = +0.124, t-test p < 0.001).
This table shows molecular features associated with OR13K1P in patient tissues and cancer cell lines. In patient samples, OR13K1P shows the broadest associations at the RNA and protein expression levels, with HNSC recurring as the lineage with the largest associated feature set.
