Q-omics provides the consensus-scored MIR4768 profile across patient tissues and cancer cell-line models. MIR4768 expression is associated with patient survival in 20 of 34 cancer types, with the highest sampling consensus in KIRC. Among the 18 cancer types available for tumor–normal comparison, MIR4768 is differentially expressed in 14, with the highest sampling consensus in KICH. Additionally, MIR4768 RNA expression shows 10,753 significant gene co-expression associations, with the highest sampling consensus in TGCT. Together, these results highlight KIRC, KICH, and TGCT as cancer lineages where MIR4768 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for MIR4768 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes MIR4768 survival associations across molecular data types. MIR4768 RNA expression shows survival associations in the most cancer types (20). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible MIR4768 RNA expression–survival associations across cancer types. High MIR4768 expression shows unfavorable associations in KIRC, ACC, LIHC and KICH, but favorable associations in LUSC and BLCA. The KIRC Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify KIRC as the clearest survival context for MIR4768 RNA expression.
This table summarizes MIR4768 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 14. The strongest signals are observed in KICH for RNA.
This table ranks reproducible tumor–normal expression differences for MIR4768. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. MIR4768 shows lower tumor expression in KICH, BRCA and KIRP and higher tumor expression in LUAD, HNSC and ESCA. The KICH box plot shows higher MIR4768 RNA expression in normal versus tumor tissue (log2 FC = −1.204, t-test p < 0.001).
This table shows molecular features associated with MIR4768 in patient tissues and cancer cell lines. In patient samples, MIR4768 shows the broadest associations at the RNA and protein expression levels, with TGCT recurring as the lineage with the largest associated feature set.
