MIR4297

RNA expression — cross-omics
Cross-omicsRNA → FUNCTION-RNAPatientPairwise association · TCGA cohorts

Across TCGA patient cohorts, MIR4297 RNA expression is significantly associated with the go_rna of many other GO terms, with 6,958 significant associations in total. KIRC shows the largest number of these associations.

The most reproducible MIR4297-associated GO terms across cancer lineages are Positive regulation of double-strand break repair via homologous recombination, Positive regulation of canonical Wnt signaling pathway, and Obsolete histone modification. Each is linked with MIR4297 in more than 24 cancer types. Because this analysis shows association rather than direction, both MIR4297-to-partner and partner-to-MIR4297 results are reported.

Each partner links to its own Q-omics profile. The box plot shows the strongest example, Positive regulation of double-strand break repair via homologous recombination grouped by MIR4297-low versus MIR4297-high in STAD.

RNA expression associated GO terms by consensus

Ranked by combined sampling and lineage consensus. X-score (MIR4297→partner) and Y-score (partner→MIR4297) are standardized regression coefficients; both directions are reported because the association is undirected. p-values are from the association test.
LineagePartner GO termX-scoreY-scorep(X)p(Y)Sampling consensusLineage consensus
STADPositive regulation of double-strand break repair via homologous recombination →+0.041+0.271<.001<.001324
STADPositive regulation of canonical Wnt signaling pathway →+0.042+0.276<.001<.001324
STADObsolete histone modification →+0.045+0.263<.001<.001324
ESCAObsolete histone methylation →+0.055+0.465<.001<.001324
STADNegative regulation of mRNA processing →+0.046+0.217<.001<.001324
STADProtein stabilization →+0.045+0.223<.001<.001324
Each partner links to its Q-omics profile. Showing the 6 strongest of 6,958 associations by consensus.

Positive regulation of double-strand break repair via homologous recombination by MIR4297 expression — STAD

Box plot of Positive regulation of double-strand break repair via homologous recombination in MIR4297-low vs MIR4297-high samples in STAD.

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Exploration