Q-omics provides the consensus-scored MIR3188 profile across patient tissues and cancer cell-line models. MIR3188 expression is associated with patient survival in 14 of 34 cancer types, with the highest sampling consensus in MESO. Among the 18 cancer types available for tumor–normal comparison, MIR3188 is differentially expressed in 5, with the highest sampling consensus in THCA. Additionally, MIR3188 RNA expression shows 6,041 significant pathway-activity associations, with the highest sampling consensus in STAD. Together, these results highlight MESO, THCA, and STAD as cancer lineages where MIR3188 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for MIR3188 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes MIR3188 survival associations across molecular data types. MIR3188 RNA expression shows survival associations in the most cancer types (14). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible MIR3188 RNA expression–survival associations across cancer types. High MIR3188 expression shows unfavorable associations in LUSC, ACC, KIRC and BLCA, but favorable associations in MESO and STAD. The MESO Kaplan–Meier curve shows clear separation, with the low-expression group declining faster, consistent with the favorable association (log-rank p = .007). Together, the overview and detailed table identify MESO as the clearest survival context for MIR3188 RNA expression.
This table summarizes MIR3188 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 5. The strongest signals are observed in THCA for RNA.
This table ranks reproducible tumor–normal expression differences for MIR3188. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. MIR3188 shows lower tumor expression in THCA, BRCA and KIRP and higher tumor expression in COAD and CHOL. The THCA box plot shows higher MIR3188 RNA expression in normal versus tumor tissue (log2 FC = −1.017, t-test p < 0.001).
This table shows molecular features associated with MIR3188 in patient tissues and cancer cell lines. In patient samples, MIR3188 shows the broadest associations at the RNA and protein expression levels, with STAD recurring as the lineage with the largest associated feature set.
