Q-omics provides the consensus-scored MIR3168 profile across patient tissues and cancer cell-line models. MIR3168 expression is associated with patient survival in 7 of 34 cancer types, with the highest sampling consensus in UVM. Among the 18 cancer types available for tumor–normal comparison, MIR3168 is differentially expressed in 3, with the highest sampling consensus in KICH. Additionally, MIR3168 RNA expression shows 6,476 significant pathway-activity associations, with the highest sampling consensus in STAD. Together, these results highlight UVM, KICH, and STAD as cancer lineages where MIR3168 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for MIR3168 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes MIR3168 survival associations across molecular data types. MIR3168 RNA expression shows survival associations in the most cancer types (7). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible MIR3168 RNA expression–survival associations across cancer types. High MIR3168 expression shows unfavorable associations in UVM, DLBC, SKCM and THYM, but favorable associations in STAD and PCPG. The UVM Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify UVM as the clearest survival context for MIR3168 RNA expression.
This table summarizes MIR3168 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 3. The strongest signals are observed in KICH for RNA.
This table ranks reproducible tumor–normal expression differences for MIR3168. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. MIR3168 shows lower tumor expression in KICH and higher tumor expression in HNSC and CHOL. The KICH box plot shows higher MIR3168 RNA expression in normal versus tumor tissue (log2 FC = −0.640, t-test p < 0.001).
This table shows molecular features associated with MIR3168 in patient tissues and cancer cell lines. In patient samples, MIR3168 shows the broadest associations at the RNA and protein expression levels, with STAD recurring as the lineage with the largest associated feature set.
