MIR155HG

Across TCGA pan-cancer cohorts, MIR155HG RNA differs between tumor and matched normal tissue in 13 of 18 cancer types tested, making tumor–normal expression one of MIR155HG’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where MIR155HG RNA is more highly expressed in tumor relative to normal tissue. In most cancer types MIR155HG is over-expressed in tumor, although a few such as KICH and LUSC show the opposite, repressed pattern.

KIRC, HNSC, and KICH are the cancer types where MIR155HG tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in MIR155HG RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	Male	IV	+1.747	<.001	11	view →
HNSC	All	All	+0.360	<.001	10	view →
KICH	Female	All	−0.854	<.001	7	view →
STAD	Female	All	+1.605	.002	4	view →
BRCA	All	All	+0.528	<.001	4	view →
LIHC	All	II,III,IV	+0.393	.004	3	view →
THCA	All	IV	+1.327	.021	2	view →
LUSC	Male	III,IV	−1.247	.006	2	view →
PAAD	Male	All	−1.070	.041	2	view →
LUAD	All	All	+0.481	.001	2	view →
PRAD	All	All	−0.348	.015	2	view →
CHOL	All	All	+0.948	.024	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 12 strongest of 13 lineages.

MIR155HG–KIRC

Open the KIRC breakdown →