Q-omics provides the consensus-scored MEMO1P5 profile across patient tissues and cancer cell-line models. MEMO1P5 expression is associated with patient survival in 12 of 34 cancer types, with the highest sampling consensus in STAD. Among the 18 cancer types available for tumor–normal comparison, MEMO1P5 is differentially expressed in 1, with the highest sampling consensus in KIRC. Additionally, MEMO1P5 RNA expression shows 7,025 significant gene co-expression associations, with the highest sampling consensus in COAD. Together, these results highlight STAD, KIRC, and COAD as cancer lineages where MEMO1P5 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for MEMO1P5 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes MEMO1P5 survival associations across molecular data types. MEMO1P5 RNA expression shows survival associations in the most cancer types (12). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible MEMO1P5 RNA expression–survival associations across cancer types. High MEMO1P5 expression shows unfavorable associations in STAD, PAAD, THCA, OV, BLCA and UCEC. The STAD Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p = .001). Together, the overview and detailed table identify STAD as the clearest survival context for MEMO1P5 RNA expression.
This table summarizes MEMO1P5 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 1. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for MEMO1P5. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. MEMO1P5 shows higher tumor expression in KIRC. The KIRC box plot shows higher MEMO1P5 RNA expression in tumor versus normal tissue (log2 FC = +0.051, t-test p = .029).
This table shows molecular features associated with MEMO1P5 in patient tissues and cancer cell lines. In patient samples, MEMO1P5 shows the broadest associations at the RNA and protein expression levels, with COAD recurring as the lineage with the largest associated feature set.
