long intergenic non-protein coding RNA 2736Genealiases: []
Q-omics provides the consensus-scored LINC02736 profile across patient tissues and cancer cell-line models. LINC02736 expression is associated with patient survival in 20 of 34 cancer types, with the highest sampling consensus in CHOL. Among the 18 cancer types available for tumor–normal comparison, LINC02736 is differentially expressed in 7, with the highest sampling consensus in THCA. Additionally, LINC02736 RNA expression shows 11,709 significant gene co-expression associations, with the highest sampling consensus in UVM. Together, these results highlight CHOL, THCA, and UVM as cancer lineages where LINC02736 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for LINC02736 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes LINC02736 survival associations across molecular data types. LINC02736 RNA expression shows survival associations in the most cancer types (20). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible LINC02736 RNA expression–survival associations across cancer types. High LINC02736 expression shows unfavorable associations in CHOL, UVM, LIHC, KIRC, BRCA and THYM. The CHOL Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify CHOL as the clearest survival context for LINC02736 RNA expression.
This table summarizes LINC02736 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 7. The strongest signals are observed in THCA for RNA.
This table ranks reproducible tumor–normal expression differences for LINC02736. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. LINC02736 shows lower tumor expression in THCA and KIRC and higher tumor expression in READ, LUAD, LUSC and KICH. The THCA box plot shows higher LINC02736 RNA expression in normal versus tumor tissue (log2 FC = −0.626, t-test p < 0.001).
This table shows molecular features associated with LINC02736 in patient tissues and cancer cell lines. In patient samples, LINC02736 shows the broadest associations at the RNA and protein expression levels, with UVM recurring as the lineage with the largest associated feature set.
