Q-omics provides the consensus-scored LINC01772 profile across patient tissues and cancer cell-line models. LINC01772 expression is associated with patient survival in 24 of 34 cancer types, with the highest sampling consensus in HNSC. Among the 18 cancer types available for tumor–normal comparison, LINC01772 is differentially expressed in 12, with the highest sampling consensus in KIRC. Additionally, LINC01772 RNA expression shows 20,300 significant gene co-expression associations, with the highest sampling consensus in UVM. Together, these results highlight HNSC, KIRC, and UVM as cancer lineages where LINC01772 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for LINC01772 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes LINC01772 survival associations across molecular data types. LINC01772 RNA expression shows survival associations in the most cancer types (24). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible LINC01772 RNA expression–survival associations across cancer types. High LINC01772 expression shows unfavorable associations in ACC, LIHC, KICH and LUSC, but favorable associations in HNSC and BRCA. The HNSC Kaplan–Meier curve shows clear separation, with the low-expression group declining faster, consistent with the favorable association (log-rank p < 0.001). Together, the overview and detailed table identify HNSC as the clearest survival context for LINC01772 RNA expression.
This table summarizes LINC01772 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 12. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for LINC01772. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. LINC01772 shows lower tumor expression in BRCA, THCA and KICH and higher tumor expression in KIRC, LIHC and HNSC. The KIRC box plot shows higher LINC01772 RNA expression in tumor versus normal tissue (log2 FC = +0.378, t-test p < 0.001).
This table shows molecular features associated with LINC01772 in patient tissues and cancer cell lines. In patient samples, LINC01772 shows the broadest associations at the RNA and protein expression levels, with UVM recurring as the lineage with the largest associated feature set.
