long intergenic non-protein coding RNA 1111Genealiases: []
Q-omics provides the consensus-scored LINC01111 profile across patient tissues and cancer cell-line models. LINC01111 expression is associated with patient survival in 15 of 34 cancer types, with the highest sampling consensus in ACC. Among the 18 cancer types available for tumor–normal comparison, LINC01111 is differentially expressed in 3, with the highest sampling consensus in KICH. Additionally, LINC01111 RNA expression shows 6,946 significant gene co-expression associations, with the highest sampling consensus in KIRP. Together, these results highlight ACC, KICH, and KIRP as cancer lineages where LINC01111 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for LINC01111 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes LINC01111 survival associations across molecular data types. LINC01111 RNA expression shows survival associations in the most cancer types (15). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible LINC01111 RNA expression–survival associations across cancer types. High LINC01111 expression shows unfavorable associations in ACC, LUAD, LGG and MESO, but favorable associations in UCS and BLCA. The ACC Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify ACC as the clearest survival context for LINC01111 RNA expression.
This table summarizes LINC01111 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 3. The strongest signals are observed in THCA for RNA.
This table ranks reproducible tumor–normal expression differences for LINC01111. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. LINC01111 shows lower tumor expression in KICH, THCA and BRCA. The KICH box plot shows higher LINC01111 RNA expression in normal versus tumor tissue (log2 FC = −1.233, t-test p < 0.001).
This table shows molecular features associated with LINC01111 in patient tissues and cancer cell lines. In patient samples, LINC01111 shows the broadest associations at the RNA and protein expression levels, with KIRP recurring as the lineage with the largest associated feature set.
