KRTAP9-2

Mutation & survival
SurvivalMutationKaplan–Meier · TCGA cohorts

Across TCGA pan-cancer cohorts, KRTAP9-2 Mutation is linked to patient survival in 2 of 34 cancer types, making it a survival-associated KRTAP9-2 data layer compared with 8 for mass-spec protein.

The strongest signal is observed in lung adenocarcinoma (LUAD), where higher KRTAP9-2 Mutation is associated with worse disease-free survival. In most high-consensus cancer types, elevated KRTAP9-2 expression acts as an unfavorable survival marker.

LUAD and LIHC are the cancer types where KRTAP9-2 Mutation most reproducibly stratifies survival.

Mutation survival associations by lineage

Ranked by sampling consensus. AUC1 and AUC2 indicate survival in the high- and low-expression groups, respectively; the lower AUC marks the poorer-surviving group. p-values are from the log-rank test.
LineageMeasureSplitStageAUC1
high
AUC2
low
pSampling consensus
LUADDFSMedianIII,IV0.1410.656<.00134view →
LIHCOSMedianAll0.2260.773.0089view →
Pink = unfavorable, green = favorable. Showing the 2 strongest of 2 lineages.

KRTAP9-2–LUAD (DFS)

Kaplan–Meier survival curve for KRTAP9-2 mutant vs wild-type samples in LUAD.

Open the LUAD breakdown →

Exploration