Across TCGA pan-cancer cohorts, KRTAP9-2 Mutation is linked to patient survival in 2 of 34 cancer types, making it a survival-associated KRTAP9-2 data layer compared with 8 for mass-spec protein.
The strongest signal is observed in lung adenocarcinoma (LUAD), where higher KRTAP9-2 Mutation is associated with worse disease-free survival. In most high-consensus cancer types, elevated KRTAP9-2 expression acts as an unfavorable survival marker.
LUAD and LIHC are the cancer types where KRTAP9-2 Mutation most reproducibly stratifies survival.