Across TCGA pan-cancer cohorts, KRTAP10-3 RNA differs between tumor and matched normal tissue in 3 of 18 cancer types tested, making tumor–normal expression one of KRTAP10-3’s most consistent transcriptional readouts.

The strongest signal is observed in stomach adenocarcinoma (STAD), where KRTAP10-3 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types KRTAP10-3 is over-expressed in tumor, although a few such as ESCA show the opposite, repressed pattern.

STAD, ESCA, and LUSC are the cancer types where KRTAP10-3 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in KRTAP10-3 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
STAD	All	All	+0.025	.009	4	view →
ESCA	Male	All	−0.042	.039	1	view →
LUSC	All	All	+0.015	.049	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 3 strongest of 3 lineages.

Exploration

Same gene, other analysis: KRTAP10-3 RNA expression & survival · all KRTAP10-3 data types