KRT32

Across TCGA pan-cancer cohorts, KRT32 Mutation is linked to patient survival in 6 of 34 cancer types, making it a survival-associated KRT32 data layer compared with 19 for mass-spec protein.

The strongest signal is observed in kidney chromophobe (KICH), where higher KRT32 Mutation is associated with worse disease-free survival. In most high-consensus cancer types, elevated KRT32 expression acts as an unfavorable survival marker.

KICH, ACC, and BLCA are the cancer types where KRT32 Mutation most reproducibly stratifies survival.

Mutation survival associations by lineage

Ranked by sampling consensus. AUC1 and AUC2 indicate survival in the high- and low-expression groups, respectively; the lower AUC marks the poorer-surviving group. p-values are from the log-rank test.

Lineage	Measure	Split	Stage	AUC1 high	AUC2 low	p	Sampling consensus
KICH	DFS	Median	All	0.025	0.904	<.001	36	view →
ACC	DFS	Median	All	0.046	0.753	<.001	36	view →
BLCA	DFS	Median	All	0.078	0.615	<.001	24	view →
LUAD	OS	Median	II,III,IV	0.171	0.750	.002	6	view →
SKCM	DFS	Median	All	0.350	0.623	.002	6	view →
COAD	OS	Median	III,IV	0.208	0.791	.008	3	view →

Pink = unfavorable, green = favorable. Showing the 6 strongest of 6 lineages.

KRT32–KICH (DFS)

Open the KICH breakdown →