Across TCGA pan-cancer cohorts, IDI2 Mutation is linked to patient survival in 2 of 34 cancer types, making it a survival-associated IDI2 data layer compared with 15 for mass-spec protein.
The strongest signal is observed in prostate adenocarcinoma (PRAD), where higher IDI2 Mutation is associated with worse disease-free survival. In most high-consensus cancer types, elevated IDI2 expression acts as an unfavorable survival marker.
PRAD and COAD are the cancer types where IDI2 Mutation most reproducibly stratifies survival.