HNRNPA1P76

Across TCGA pan-cancer cohorts, HNRNPA1P76 RNA differs between tumor and matched normal tissue in 6 of 18 cancer types tested, making tumor–normal expression one of HNRNPA1P76’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where HNRNPA1P76 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types HNRNPA1P76 is over-expressed in tumor, although a few such as UCEC and COAD show the opposite, repressed pattern.

KIRC, KICH, and LIHC are the cancer types where HNRNPA1P76 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in HNRNPA1P76 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	All	II,III,IV	+0.039	<.001	11	view →
KICH	All	IV	+0.072	<.001	4	view →
LIHC	Female	All	+0.051	.004	3	view →
UCEC	All	IV	−0.215	.004	2	view →
KIRP	Female	All	+0.068	.024	2	view →
COAD	All	II,III,IV	−0.039	.029	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 6 strongest of 6 lineages.

HNRNPA1P76–KIRC

Open the KIRC breakdown →