HMGN1P11

Across TCGA pan-cancer cohorts, HMGN1P11 RNA differs between tumor and matched normal tissue in 2 of 18 cancer types tested, making tumor–normal expression one of HMGN1P11’s most consistent transcriptional readouts.

The strongest signal is observed in lung adenocarcinoma (LUAD), where HMGN1P11 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types HMGN1P11 is over-expressed in tumor, although a few such as KICH show the opposite, repressed pattern.

LUAD and KICH are the cancer types where HMGN1P11 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in HMGN1P11 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
LUAD	Female	II,III,IV	+0.169	.001	3	view →
KICH	All	All	−0.055	.029	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 2 strongest of 2 lineages.

HMGN1P11–LUAD

Open the LUAD breakdown →