Across TCGA pan-cancer cohorts, FTH1P24 RNA differs between tumor and matched normal tissue in 2 of 18 cancer types tested, making tumor–normal expression one of FTH1P24’s most consistent transcriptional readouts.
The strongest signal is observed in prostate adenocarcinoma (PRAD), where FTH1P24 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types FTH1P24 is over-expressed in tumor.
PRAD and KIRC are the cancer types where FTH1P24 tumor–normal differential expression is most reproducible.