Q-omics provides the consensus-scored FTH1P20 profile across patient tissues and cancer cell-line models. FTH1P20 expression is associated with patient survival in 22 of 34 cancer types, with the highest sampling consensus in UVM. Among the 18 cancer types available for tumor–normal comparison, FTH1P20 is differentially expressed in 12, with the highest sampling consensus in KIRC. Additionally, FTH1P20 RNA expression shows 16,513 significant gene co-expression associations, with the highest sampling consensus in ACC. Together, these results highlight UVM, KIRC, and ACC as cancer lineages where FTH1P20 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for FTH1P20 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes FTH1P20 survival associations across molecular data types. FTH1P20 RNA expression shows survival associations in the most cancer types (22). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible FTH1P20 RNA expression–survival associations across cancer types. High FTH1P20 expression shows unfavorable associations in UVM, KICH, HNSC, LIHC, UCEC and LUAD. The UVM Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify UVM as the clearest survival context for FTH1P20 RNA expression.
This table summarizes FTH1P20 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 12. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for FTH1P20. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. FTH1P20 shows lower tumor expression in COAD and higher tumor expression in KIRC, KIRP, THCA, LIHC and HNSC. The KIRC box plot shows higher FTH1P20 RNA expression in tumor versus normal tissue (log2 FC = +1.317, t-test p < 0.001).
This table shows molecular features associated with FTH1P20 in patient tissues and cancer cell lines. In patient samples, FTH1P20 shows the broadest associations at the RNA and protein expression levels, with ACC recurring as the lineage with the largest associated feature set.
