ELMOD1

Across TCGA pan-cancer cohorts, ELMOD1 RNA differs between tumor and matched normal tissue in 13 of 18 cancer types tested, making tumor–normal expression one of ELMOD1’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where ELMOD1 RNA is repressed in tumor relative to normal tissue. In most cancer types ELMOD1 is over-expressed in tumor, although a few such as KIRC and KIRP show the opposite, repressed pattern.

KIRC, KIRP, and UCEC are the cancer types where ELMOD1 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in ELMOD1 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	Male	All	−0.704	<.001	9	view →
KIRP	Male	III,IV	−1.060	<.001	8	view →
UCEC	All	All	−0.550	<.001	8	view →
LUAD	Female	III,IV	−0.632	<.001	7	view →
LUSC	Male	II,III,IV	−0.398	<.001	7	view →
STAD	All	All	−0.139	.004	5	view →
THCA	Male	All	−0.106	.012	3	view →
READ	Female	All	−0.322	.007	2	view →
PRAD	All	All	−0.243	<.001	2	view →
PAAD	Male	All	−0.175	.013	2	view →
COAD	Male	All	−0.139	<.001	2	view →
BRCA	Female	II,III,IV	+0.092	.015	2	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 12 strongest of 13 lineages.

ELMOD1–KIRC

Open the KIRC breakdown →