DIPK2B

Across TCGA cell cohorts, DIPK2B RNA expression is significantly associated with the go_rna of many other GO terms, with 316 significant associations in total. SKIN shows the largest number of these associations.

The most reproducible DIPK2B-associated GO terms across cancer lineages are Regulation of cellular response to vascular endothelial growth factor stimulus, tRNA threonylcarbamoyladenosine modification, and Hormone-mediated signaling pathway. Each is linked with DIPK2B in more than 2 cancer types. Because this analysis shows association rather than direction, both DIPK2B-to-partner and partner-to-DIPK2B results are reported.

Each partner links to its own Q-omics profile. The box plot shows the strongest example, Regulation of cellular response to vascular endothelial growth factor stimulus grouped by DIPK2B-low versus DIPK2B-high in BREAST.

RNA expression associated GO terms by consensus

Ranked by combined sampling and lineage consensus. X-score (DIPK2B→partner) and Y-score (partner→DIPK2B) are standardized regression coefficients; both directions are reported because the association is undirected. p-values are from the association test.

Lineage	Partner GO term	X-score	Y-score	p(X)	p(Y)	Sampling consensus	Lineage consensus
BREAST	Regulation of cellular response to vascular endothelial growth factor stimulus →	+0.094	+0.008	.004	.001	3	3
LUNG_SCLC	tRNA threonylcarbamoyladenosine modification →	-0.113	-0.010	<.001	.002	3	3
LUNG_SCLC	Hormone-mediated signaling pathway →	+0.058	+0.009	.004	.008	3	3
BREAST	Regulation of receptor clustering →	+0.111	+0.007	<.001	.008	3	2
BREAST	Cellular response to vascular endothelial growth factor stimulus →	+0.111	+0.012	.001	.002	3	2
BREAST	Ectodermal placode formation →	+0.099	+0.010	<.001	<.001	3	2

Each partner links to its Q-omics profile. Showing the 6 strongest of 316 associations by consensus.

Regulation of cellular response to vascular endothelial growth factor stimulus by DIPK2B expression — BREAST

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