CSNK1G2-AS1

Across TCGA pan-cancer cohorts, CSNK1G2-AS1 RNA differs between tumor and matched normal tissue in 8 of 18 cancer types tested, making tumor–normal expression one of CSNK1G2-AS1’s most consistent transcriptional readouts.

The strongest signal is observed in colon adenocarcinoma (COAD), where CSNK1G2-AS1 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types CSNK1G2-AS1 is over-expressed in tumor, although a few such as THCA show the opposite, repressed pattern.

COAD, KIRC, and CHOL are the cancer types where CSNK1G2-AS1 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in CSNK1G2-AS1 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
COAD	All	IV	+0.474	<.001	11	view →
KIRC	All	All	+0.087	<.001	9	view →
CHOL	Male	All	+0.276	.003	4	view →
STAD	All	II,III,IV	+0.192	.001	4	view →
BRCA	All	All	+0.095	.035	2	view →
THCA	Female	II,III,IV	−0.071	.010	2	view →
READ	All	All	+0.210	.017	1	view →
KIRP	Female	All	+0.050	.046	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 8 strongest of 8 lineages.

CSNK1G2-AS1–COAD

Open the COAD breakdown →