CNTNAP3P1

Across TCGA pan-cancer cohorts, CNTNAP3P1 RNA differs between tumor and matched normal tissue in 9 of 18 cancer types tested, making tumor–normal expression one of CNTNAP3P1’s most consistent transcriptional readouts.

The strongest signal is observed in kidney chromophobe (KICH), where CNTNAP3P1 RNA is repressed in tumor relative to normal tissue. In most cancer types CNTNAP3P1 is over-expressed in tumor, although a few such as KICH and KIRC show the opposite, repressed pattern.

KICH, KIRC, and LUSC are the cancer types where CNTNAP3P1 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in CNTNAP3P1 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KICH	All	All	−0.154	<.001	8	view →
KIRC	All	II,III,IV	−0.105	.001	8	view →
LUSC	Female	All	−0.130	<.001	7	view →
LUAD	All	All	−0.091	.001	7	view →
THCA	All	All	−0.172	<.001	6	view →
BRCA	Female	II,III,IV	−0.111	<.001	6	view →
KIRP	All	All	−0.118	.001	5	view →
BLCA	Female	All	−0.068	.001	4	view →
UCEC	All	All	−0.144	.005	2	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 9 strongest of 9 lineages.

CNTNAP3P1–KICH

Open the KICH breakdown →