CHORDC1P4

Across TCGA pan-cancer cohorts, CHORDC1P4 RNA differs between tumor and matched normal tissue in 9 of 18 cancer types tested, making tumor–normal expression one of CHORDC1P4’s most consistent transcriptional readouts.

The strongest signal is observed in stomach adenocarcinoma (STAD), where CHORDC1P4 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types CHORDC1P4 is over-expressed in tumor, although a few such as KIRP and KIRC show the opposite, repressed pattern.

STAD, HNSC, and KIRP are the cancer types where CHORDC1P4 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in CHORDC1P4 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
STAD	All	All	+0.395	<.001	8	view →
HNSC	Male	II,III,IV	+0.101	.001	7	view →
KIRP	Male	All	−0.130	.005	5	view →
BRCA	All	All	+0.120	.030	4	view →
KIRC	All	II,III,IV	−0.067	.015	3	view →
ESCA	Male	All	+0.863	.006	2	view →
PRAD	All	All	+0.138	.026	2	view →
KICH	All	All	−0.107	.007	2	view →
THCA	Female	All	−0.086	.003	2	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 9 strongest of 9 lineages.

CHORDC1P4–STAD

Open the STAD breakdown →