CFAP52

Across TCGA pan-cancer cohorts, CFAP52 RNA differs between tumor and matched normal tissue in 13 of 18 cancer types tested, making tumor–normal expression one of CFAP52’s most consistent transcriptional readouts.

The strongest signal is observed in kidney chromophobe (KICH), where CFAP52 RNA is repressed in tumor relative to normal tissue. In most cancer types CFAP52 is over-expressed in tumor, although a few such as KICH and LUAD show the opposite, repressed pattern.

KICH, LUAD, and LUSC are the cancer types where CFAP52 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in CFAP52 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KICH	Male	All	−0.513	<.001	11	view →
LUAD	All	III,IV	−1.840	<.001	9	view →
LUSC	Female	II,III,IV	−2.172	<.001	8	view →
THCA	Male	All	−0.388	<.001	8	view →
BLCA	All	All	+0.303	<.001	8	view →
KIRP	All	All	+0.320	.002	6	view →
STAD	Male	All	+0.187	.002	4	view →
CHOL	All	All	+0.523	<.001	3	view →
COAD	Male	III,IV	+0.402	.026	3	view →
UCEC	All	All	−1.267	.041	2	view →
PAAD	Female	All	+0.874	.014	2	view →
PRAD	All	All	+0.204	.002	2	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 12 strongest of 13 lineages.

CFAP52–KICH

Open the KICH breakdown →