CCDC97

Across TCGA cell cohorts, CCDC97 RNA expression is significantly associated with the go_rna of many other GO terms, with 3,796 significant associations in total. LARGE_INTESTINE shows the largest number of these associations.

The most reproducible CCDC97-associated GO terms across cancer lineages are Negative regulation of DNA-templated transcription, elongation, Negative regulation of calcium ion export across plasma membrane, and Positive regulation of T-helper 17 cell lineage commitment. Each is linked with CCDC97 in more than 16 cancer types. Because this analysis shows association rather than direction, both CCDC97-to-partner and partner-to-CCDC97 results are reported.

Each partner links to its own Q-omics profile. The box plot shows the strongest example, Negative regulation of DNA-templated transcription, elongation grouped by CCDC97-low versus CCDC97-high in STOMACH.

RNA expression associated GO terms by consensus

Ranked by combined sampling and lineage consensus. X-score (CCDC97→partner) and Y-score (partner→CCDC97) are standardized regression coefficients; both directions are reported because the association is undirected. p-values are from the association test.

Lineage	Partner GO term	X-score	Y-score	p(X)	p(Y)	Sampling consensus	Lineage consensus
STOMACH	Negative regulation of DNA-templated transcription, elongation →	+0.098	+0.808	.005	.004	3	17
UPPER_AERODIGESTIVE_TRACT	Negative regulation of calcium ion export across plasma membrane →	+0.061	+0.566	<.001	<.001	3	16
STOMACH	Positive regulation of T-helper 17 cell lineage commitment →	+0.253	+1.180	.002	.001	3	16
STOMACH	Positive regulation of gene expression, epigenetic →	+0.087	+1.111	.001	<.001	3	16
BONE	mRNA alternative polyadenylation →	+0.077	+0.591	<.001	.002	3	15
BONE	Epigenetic regulation of gene expression →	+0.094	+0.626	<.001	.005	3	15

Each partner links to its Q-omics profile. Showing the 6 strongest of 3,796 associations by consensus.

Negative regulation of DNA-templated transcription, elongation by CCDC97 expression — STOMACH

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