CCDC102A

Mutation & survival
SurvivalMutationKaplan–Meier · TCGA cohorts

Across TCGA pan-cancer cohorts, CCDC102A Mutation is linked to patient survival in 3 of 34 cancer types, making it a survival-associated CCDC102A data layer compared with 24 for mass-spec protein and 6 for mass-spec protein.

The strongest signal is observed in stomach adenocarcinoma (STAD), where higher CCDC102A Mutation is associated with worse overall survival. In most high-consensus cancer types, elevated CCDC102A expression acts as an unfavorable survival marker.

STAD, LIHC, and UCEC are the cancer types where CCDC102A Mutation most reproducibly stratifies survival.

Mutation survival associations by lineage

Ranked by sampling consensus. AUC1 and AUC2 indicate survival in the high- and low-expression groups, respectively; the lower AUC marks the poorer-surviving group. p-values are from the log-rank test.
LineageMeasureSplitStageAUC1
high
AUC2
low
pSampling consensus
STADOSMedianAll0.0930.690<.00136view →
LIHCOSMedianAll0.0410.781<.00136view →
UCECOSMedianIV0.2310.592.0366view →
Pink = unfavorable, green = favorable. Showing the 3 strongest of 3 lineages.

CCDC102A–STAD (OS)

Kaplan–Meier survival curve for CCDC102A mutant vs wild-type samples in STAD.

Open the STAD breakdown →

Exploration