CAMTA1

Across TCGA pan-cancer cohorts, CAMTA1 RNA differs between tumor and matched normal tissue in 10 of 18 cancer types tested, making tumor–normal expression one of CAMTA1’s most consistent transcriptional readouts.

The strongest signal is observed in kidney chromophobe (KICH), where CAMTA1 RNA is repressed in tumor relative to normal tissue. In most cancer types CAMTA1 is over-expressed in tumor, although a few such as KICH and KIRP show the opposite, repressed pattern.

KICH, HNSC, and KIRP are the cancer types where CAMTA1 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in CAMTA1 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KICH	Female	II,III,IV	−1.084	<.001	10	view →
HNSC	Male	III,IV	+0.503	.004	6	view →
KIRP	All	All	−0.293	.001	6	view →
LIHC	Male	II,III,IV	+0.564	<.001	5	view →
KIRC	Male	III,IV	−0.224	.025	5	view →
CHOL	Male	All	+0.745	<.001	3	view →
PRAD	All	All	−0.221	<.001	2	view →
BRCA	Female	All	−0.139	.037	2	view →
BLCA	Male	IV	−0.722	.041	1	view →
LUSC	Male	III,IV	−0.512	.042	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 10 strongest of 10 lineages.

CAMTA1–KICH

Open the KICH breakdown →