CAMTA1-DT

Across TCGA pan-cancer cohorts, CAMTA1-DT RNA differs between tumor and matched normal tissue in 13 of 18 cancer types tested, making tumor–normal expression one of CAMTA1-DT’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where CAMTA1-DT RNA is repressed in tumor relative to normal tissue. In most cancer types CAMTA1-DT is over-expressed in tumor, although a few such as KIRC and KICH show the opposite, repressed pattern.

KIRC, KICH, and THCA are the cancer types where CAMTA1-DT tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in CAMTA1-DT RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	Male	II,III,IV	−1.265	<.001	10	view →
KICH	Male	All	−1.657	<.001	8	view →
THCA	Male	IV	−1.453	<.001	8	view →
STAD	All	All	+0.419	.001	6	view →
BLCA	Female	III,IV	+0.815	.013	5	view →
KIRP	All	All	−0.615	.004	5	view →
LUSC	All	All	+0.520	<.001	4	view →
BRCA	All	II,III,IV	−0.199	.010	4	view →
LIHC	All	All	+0.295	.004	3	view →
ESCA	All	II,III,IV	+1.505	.040	2	view →
CHOL	All	All	+0.592	.002	2	view →
LUAD	All	All	+0.287	.014	2	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 12 strongest of 13 lineages.

CAMTA1-DT–KIRC

Open the KIRC breakdown →