Across TCGA pan-cancer cohorts, C1orf162 Mutation is linked to patient survival in 3 of 34 cancer types, making it a survival-associated C1orf162 data layer compared with 21 for mass-spec protein and 1 for mass-spec protein.
The strongest signal is observed in sarcoma (SARC), where higher C1orf162 Mutation is associated with worse disease-free survival. In most high-consensus cancer types, elevated C1orf162 expression acts as an unfavorable survival marker.
SARC, LUSC, and SKCM are the cancer types where C1orf162 Mutation most reproducibly stratifies survival.